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Table 1 Experimental variables

From: Modulation of mitochondrial function with near-infrared light reduces brain injury in a translational model of cardiac arrest

  

Sham

Cardiac arrest

Cardiac arrest + NIR

pH

Baseline

7.47 ± 0.04

7.49 ± 0.05

7.49 ± 0.05 (NS)

 

30 min

7.48 ± 0.05

7.29 ± 0.09

7.33 ± 0.10 (NS)

PCO2 (mmHg)

Baseline

35.1 ± 4.10

37.15 ± 4.20

36.5 ± 4.20 (NS)

 

30 min

36.28 ± 4.30

39.34 ± 12.80

42.65 ± 2.60 (NS)

PO2 (mmHg)

Baseline

362.6 ± 65.10

383.6 ± 72.20

394.9 ± 80.20 (NS)

 

30 min

358.2 ± 26.60

318.0 ± 103.30

406.3 ± 53.90 (NS)

HCO3- (mmol/L)

Baseline

25.67 ± 4.60

28.3 ± 3.40

28.44 ± 6.60 (NS)

 

30 min

26.75 ± 2.50

21.0 ± 6.40

22.4 ± 4.00 (NS)

Lactate (mmol/L)

Baseline

0.862 ± 0.20

0.9 ± 0.55

0.68 ± 0.30 (NS)

 

30 min

0.91 ± 0.30

4.97 ± 2.00

5.55 ± 1.30 (NS)

CPR etCO2 (mmHg)

  

18.20 ± 7.63

25.71 ± 8.81 (NS)

Time to ROSC (min)

  

12.14 ± 1.40

11.84 ± 1.00 (NS)

  1. Blood gas parameters measured from fresh arterial blood with an i-STAT handheld blood analyzer at baseline (before arrest) and at 30 post-ROSC. Comparison of experimental variables was made with one-way ANOVA, and stepwise multiple comparisons were made between groups with Newman–Keuls post hoc test. Changes in pH, HCO3 (bicarbonate) and Lactate were observed between baseline and 30 min post-ROSC within groups, as compared by one-way ANOVA repeated measures with mixed effects. Values are reported as mean ± SD. Comparisons were made between cardiac arrest and cardiac arrest + NIR at each time point and p < 0.05 was considered significant. NS Not significant
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Critical Care

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