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Figure 4 | Critical Care

Figure 4

From: Early propranolol treatment induces lung heme-oxygenase-1, attenuates metabolic dysfunction, and improves survival following experimental sepsis

Figure 4

Heat shock protein (HSP) expression in propranolol and control groups. Mean relative densitometry of all HSPs normalized to beta-actin. No changes in HSP expression were noted, with the exception of HO-1 expression. There were similar levels of HO-1 between groups at 6 hours. At 24 hours post sepsis onset propranolol treated rats had significantly higher levels of HO-1 compared to saline treated rats (*-P < 0.05). Error bars represent standard deviation. Image representative of HSP expression and beta-actin from the lungs in saline and propranolol groups at 6 and 24 hours. Error bars represent standard deviation (n = 4/group).

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