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  • Open Access

Impact of an open lung approach on hemodynamic parameters after cardiac surgery

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Critical Care201216 (Suppl 1) :P123

  • Published:


  • Hemodynamic Parameter
  • Inspiratory Pressure
  • Arterial Oxygen Saturation
  • Conventional Group
  • Respiratory Mechanic


Lung recruitment maneuver (RM) has been associated with an increase of arterial oxygen saturation and improvement of respiratory parameters. Nevertheless, adverse hemodynamic effects can occur due to the RM technique. The aim of this study is to evaluate the effect of the RM on hemodynamic parameters in the immediate postoperative period after cardiac surgery.


A total of 120 patients with PaO2/FiO2 ratio <250 was randomized to a conventional strategy of mechanical ventilation or open lung strategy. The open lung strategy was performed using RM with an inspiratory pressure amplitude of 15 cmH2O and PEEP of 30 cmH2O three times during 1 minute and setting PEEP after RM at 13 cmH2O. The conventional strategy was done using PEEP = 8 cmH2O and RM with CPAP = 20 cmH2O three times during 30 seconds and setting PEEP after RM at 8 cmH2O. The heart rate, systolic, diastolic and mean arterial blood pressures were recorded before, immediately and 5 minutes after RM. Respiratory mechanics and blood gas analysis were recorded before and after RM.


The open lung group presented a higher variability on blood pressure immediately after RM compared to the conventional group. There were no differences in baseline blood pressure or 5 minutes after RM and heart rate between groups. The open lung group presented higher lung compliance (60 ± 17 vs. 48 ± 13 ml/cmH2O) and PaO2/FiO2 (431 ± 124 vs. 229 ± 68) ratio compared to the conventional group.


An open lung approach after cardiac surgery improves lung compliance and the PaO2/FiO2 ratio with minimum hemodynamic detrimental effect.

Authors’ Affiliations

Heart Institute, São Paulo, Brazil


  1. Amato MB, Barbas CS, Medeiros DM, et al.: Effect of a protective-ventilation strategy on mortality in the acute respiratory distress syndrome. N Engl J Med 1998, 338: 347-354. 10.1056/NEJM199802053380602View ArticlePubMedGoogle Scholar


© Leme et al.; licensee BioMed Central Ltd. 2012

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.