Fig. 3From: Association of deranged cerebrovascular reactivity with brain injury following cardiac arrest: a post-hoc analysis of the COMACARE triala–c 48 h neuron-specific enolase (NSE) concentrations (\(\mathrm{\mu g}/\mathrm{l}\)), d–f 48 h S100B concentrations (\(\mathrm{\mu g}/\mathrm{l}\)) and g–i 48 h neurofilament (NfL) concentrations (pg/ml) against mean cerebrovascular reactivity (mean TOx) during the time intervals 0–12 h, 12–24 h and 24–48 h. Patients are classified according to good or poor neurologic outcomes. Data is presented with logharitmic X-axesBack to article page