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Table 2 Oxygenation, patient temperature and haemodynamics at the time of measurement

From: Non-pulsatile blood flow is associated with enhanced cerebrovascular carbon dioxide reactivity and an attenuated relationship between cerebral blood flow and regional brain oxygenation

Flow mode PaCO2 levelrSO2 (%)SaO2 (%)PaO2 (mmHg)Temp (°C)MAP (mmHg)CI (L/min/m2)
Non-pulsatile
 30 mmHg56 [51–60]99 [98–100]160 [140–177]36.3 [35.9–36.7]63 [57–68]2.5 [2.4–2.7]
 40 mmHg59 [57–63]99 [98–100]170 [154–197]36.4 [35.8–36.7]63 [57–69]2.5 [2.4–2.7]
 50 mmHg62 [57–66]99 [98–100]161 [133–190]36.4 [35.9–36.7]63 [54–68]2.5 [2.3–2.7]
Pulsatile
 30 mmHg58 [56–64]100* [99–100]137* [116–161]36.5 [36.3–36.7]71* [66–80]2.7 [2.1–3.3]
 40 mmHg61 [59–67]100* [100–100]140* [109–164]36.4 [36.3–36.7]72* [65–83]2.6 [1.9–2.8]
 50 mmHg64 [62–70]100* [99–100]120* [101–146]36.5 [36.3–36.7]68* [65–75]2.6 [2.0–3.3]
  1. Values are medians [25th–75th percentile], rSO2 Regional brain saturation, SaO2 Arterial haemoglobin oxygen saturation, PaO2 Arterial partial pressure of oxygen, Temp patient temperature MAP mean arterial blood pressure, CI cardiac index (n = 8), * = p < 0.05 vs. corresponding value during non-pulsatile flow (ANOVA with post hoc Tukey test)