Fig. 2From: Exogenous vasopressin dose-dependently modulates gastric microcirculatory oxygenation in dogs via V1A receptorGastric microcirculation. Gastric microcirculatory oxygenation (μHbO2, a) and gastric microcirculatory flow (μflow, b) in anesthetized dogs in time control experiment (C), with sole V1A receptor blockade (VB) and with AVP dose escalation (dose 1, 0.001 ng/kg/min; dose 2, 0.01 ng/kg/min; dose 3, 0.1 ng/kg/min; dose 4, 1 ng/kg/min) with (AVP VB) or without (AVP) V1A receptor blockade. Data are presented as individual values + mean for n = 6 dogs; *p < 0.05 vs. baseline; #p < 0.05 vs. control treatment (C), 2-way ANOVA for repeated measurements followed by Dunnett’s post hoc test; §p < 0.05 for AVP VB vs. AVP, 2-way ANOVA for repeated measurements followed by Bonferroni’s post hoc testBack to article page