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Fig. 3 | Critical Care

Fig. 3

From: Inhaled nitric oxide improves transpulmonary blood flow and clinical outcomes after prolonged cardiac arrest: a large animal study

Fig. 3

Inhaled nitric oxide (iNO) improves cerebral oxygenation and suppresses the increase of glucose after return of spontaneous circulation (ROSC). Comparison of animals treated with 20 ppm iNO (n = 10) or Control (n = 10). Mean ± standard error of the mean: *p ≤0.05, comparing iNO to Control. BL baseline (i.e., 5 minutes prior to cardiac arrest). a Cerebral oxygenation (CeSO2) was estimated using transcutaneous spectroscopy using near-infrared light. Due to limited availability during experiments, values were estimated in 6 animals per group. b Serum glucose levels before and after cardiac arrest and resuscitation

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