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Fig. 2 | Critical Care

Fig. 2

From: Inhaled nitric oxide improves transpulmonary blood flow and clinical outcomes after prolonged cardiac arrest: a large animal study

Fig. 2

Inhaled nitric oxide (iNO) impacts hemodynamics during and after cardiac arrest. Comparison of animals treated with 20 ppm iNO (n = 10) or Control (n = 10). Mean ± standard error of the mean: *p ≤0.05, comparing iNO to Control. BL baseline (i.e., 5 minutes prior to cardiac arrest), CPR cardiopulmonary resuscitation. a Pump flow of the intravascular cardiopulmonary resuscitation device. b Calculated coronary perfusion pressure (CPP). c Mean pulmonary artery pressure (MPAP) measured at BL and 1, 2, 3, 4, 5, and 6 hours after return of spontaneous circulation (ROSC). d Calculated pulmonary vascular resistance (PVR) before (BL), during (1–6 minutes), and following cardiac arrest and resuscitation (7 minutes). Time indicates period since start of CPR. The 10-minute period of untreated ventricular fibrillation preceding CPR was omitted for clarity

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Critical Care

ISSN: 1364-8535