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Table 2 Echocardiographic data

From: Hypervolemia induces and potentiates lung damage after recruitment maneuver in a model of sepsis-induced acute lung injury

   HYPO NORMO HYPER
   NR RM-CPAP NR RM-CPAP NR RM-CPAP
Cardiac
Output (ml.min-1)
BASELINE 20 ± 10 20 ± 10 20 ± 10 20 ± 10 20 ± 10 40 ± 10†§
  END 10 ± 10 10 ± 10 10 ± 10 20 ± 10 60 ± 10* **# 60 ± 10‡§
Stroke volume (ml) BASELINE 0.17 ± 0.01 0.13 ± 0.01† 0.13 ± 0.01** 0.13 ± 0.01 0.10 ± 0.05** 0.13 ± 0.01
  END 0.10 ± 0.01* 0.10 ± 0.01 0.10 ± 0.01 0.13 ± 0.01 0.33 ± 0.01**# 0.26 ± 0.01*†‡§
Ejection
fraction (%)
BASELINE 74 ± 1 73 ± 3 78 ± 4 74 ± 4 74 ± 1 68 ± 7
  END 63 ± 4* 65 ± 1* 71 ± 1 73 ± 1‡ 86 ± 3* **# 88 ± 3*‡§
  1. Echocardiographic data measured at BASELINE and after one hour of mechanical ventilation (END). Animals were randomly assigned to hypovolemia (HYPO), normovolemia (NORMO) or hypervolemia (HYPER) with recruitment maneuver (RM-CPAP) or not (NR). Values are mean ± standard error of the mean of six rats in each group. *Significantly different from BASELINE (P < 0.05). †Significantly different from NR (P < 0.05). **Significantly different from HYPO-NR (P < 0.05). ‡Significantly different from HYPO-RM-CPAP (P < 0.05). #Significantly different from NORMO-NR (P < 0.05). §Significantly different from NORMO-RM-CPAP (P < 0.05).