Table 3 Rodent studies
From: The histopathology of septic acute kidney injury: a systematic review
Study | Induction of sepsis | Acute tubular necrosis |
|---|---|---|
Hurley and colleagues [32] | Salmonella enteritidis endotoxin | No |
Miyaji and colleagues [33] | Cecal ligation perforation/lipopolysaccharide | Yes |
Sato and colleagues [34] | Escherichia coli | No |
Hayashi and colleagues [35] | Lipopolysaccharide-induced sepsis | No |
Tsao and colleagues [36] | Lipopolysaccharide-induced sepsis | No |
Kadkhodaee and Qasemi [37] | Lipopolysaccharide-induced sepsis | Yes |
Zager and Prior [38] | E. coli septicemia | No |
Wang and colleagues [39] | Lipopolysaccharide-induced sepsis | Yes |
Tiwari and colleagues [40] | Lipopolysaccharide-induced sepsis | No |
Gallos and colleagues [41] | Cecal ligation perforation | No |
Kikeri and colleagues [42] | Lipopolysaccharide-induced sepsis | No |
Yokota and colleagues [43] | Lipopolysaccharide-induced sepsis | No |