Figure 4From: Neutrophils from critically ill septic patients mediate profound loss of endothelial barrier integrityNeutrophils from acute respiratory distress syndrome patients induce greater endothelial barrier dysfunction. Change in monolayer resistance at 30 minutes, 60 minutes, and 180 minutes. Groups were analyzed using two-way analysis of variance with Holm–Sidak post-hoc analysis. Values presented as mean ± standard error of the mean. Neutrophils from septic patients with acute respiratory distress syndrome (ARDS) produced greater loss of barrier function than those obtained from septic patients without ARDS. *P <0.05.Back to article page