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Figure 7 | Critical Care

Figure 7

From: Oxygen deficit and H2S in hemorrhagic shock in rats

Figure 7

Ability of plasma and tissue homogenates to oxidize/complex H 2 S. Individual relationship between vitamin B12 concentration and residual H2S concentration ([H2S]r) determined two minutes after addition of 100 μM H2S in the plasma (Panel A), in the supernatant of kidney homogenates (Panel B) and heart homogenates (Panel C) in all the control (open circles) or vitamin B12-treated (closed circles) rats. [H2S]r is expressed in percentage of the initial concentration of 100 μM present in a control PBS solution analyzed at the same time as the medium. In the presence of vitamin B12, very large amounts of H2S can be oxidized both in the plasma and in the kidney. In the heart, the concentration of vitamin B12 was too low to be measured by our method (except for two rats) and no visible effect on high μM concentration of exogenous H2S could be identified (see text for additional comments).

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