Table 1 Clinical summary of quantitative EEG in subarachnoid hemorrhage

Labar [7]

HH I: 2

1) Focal neurological deficit

Ischemic events (n = 18)

5 silent infarcts detected by qEEG alone

HH III: 8

2) Global cortical dysfunction

4 qEEG changes prior to clinical changes

HH IV: 1

3) Encephalopathy

Vespa [8]

Awake

(HH I-III)

1) Angiographic vasospasm

Vasospasm (n = 19)

All vasospasm with qEEG changes.

2) TCD vasospasm (> 120 cm/s or Lindegaard ratio > 3)

10/19 qEEG changed mean 2.9 days prior to vasospasm confirmation.

Claassen [9]

Comatose

(HH IV-V)

DCI

DCI (n = 9)

Raw EEG changed in 78%; qEEG sensitive to a 10% change in 6 post-stimulation minutes or 50% change in only 1 post-stimulation minute.

1) Clinical deterioration

2) New infarct on CT

Rathakrishnan [10]

mF 3-4

DCI

DCI (n = 8)

qEEG sensitivity with clinical data is 67%.

HH I: 1

1) Clinical deterioration

3/8 qEEG changed more than 24 hours prior to clinical change.

HH II: 5

2) New infarct on CT

HH III: 2

HH IV: 3

HH V: 1